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Neuropsychological Aspects Of Post Traumatic Stress Disorder

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Running head: POST-TRAUMATIC STRESS DISORDER 1
The Neuropsychological Aspects of Post-Traumatic Stress Disorder

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POST-TRAUMATIC STRESS DISORDER 2
The purpose of the present paper is to discuss the neuropsychological factors that explain
post-traumatic stress disorder (PTSD) in terms of etiology, pathology, treatment option, and
diagnostic. I have decided to review PTSD because it is a disorder that affects the lives of
millions of people and I believe it is important to gain a better understanding of it.
PTSD has a complex etiology, as there are several factors that can contribute to the
development of it, including trauma, negative childhood experiences, genetical predisposition,
and substance abuse. That being said, the neurobiology of PTSD remains unknown and there is
no single neuropsychological theory of PTSD that can explain it. At the present time, most
researchers agree that people with PTSD have increased levels of corticotropin-releasing
hormone, lower cortisol at the basal level, and increased negative feedback suppression of the
hypothalamicpituitaryadrenal axis by dexamethasone (Scott et al., 2014). Traumatic events are
a fundamental factor in the development of PTSD. For instance, traumatic events can often cause
PTSD even in people who do not have a genetic predisposition toward it. Common traumatic
events that lead to PTSD include sexual assault and war experience. Negative childhood
experiences are known to correlate with PTSD, including domestic violence and foster care.
Evidence also shows that susceptibility to PTSD is hereditary, and around 30% of the variance in
PTSD is likely caused by genetics. For instance, evidence shows that people with a smaller
hippocampus are more likely to suffer from PTSD after having been exposed to a traumatic
event (Yurgil et al., 2014). Finally, substance abuse often co-occurs with PTSD, but the exact
relationship between the two disorder is difficult to determine; in some cases, substance abusers
may be more prone toward experiencing a traumatic event while in other cases substance abuse
might predispose individuals from developing PTSD after a traumatic event. More so, some
genes that correlate with PTSD also correlate with substance abuse (Wrocklage et al., 2016).

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Running head: POST-TRAUMATIC STRESS DISORDER The Neuropsychological Aspects of Post-Traumatic Stress Disorder 1 POST-TRAUMATIC STRESS DISORDER 2 The purpose of the present paper is to discuss the neuropsychological factors that explain post-traumatic stress disorder (PTSD) in terms of etiology, pathology, treatment option, and diagnostic. I have decided to review PTSD because it is a disorder that affects the lives of millions of people and I believe it is important to gain a better understanding of it. PTSD has a complex etiology, as there are several factors that can contribute to the development of it, including trauma, negative childhood experiences, genetical predisposition, and substance abuse. That being said, the neurobiology of PTSD remains unknown and there is no single neuropsychological theory of PTSD that can explain it. At the present time, most researchers agree that people with PTSD have increased levels of corticotropin-releasing hormone, lower cortisol at the basal level, and increased negative feedback suppression of the hypothalamic–pituitary–adrenal axis by dexamethasone (Scott et al., 2014). Traumatic events are a fundamental factor in the development of PTSD. For instance, traumatic events can often cause PTSD even in people who do not have a genetic predisposition toward it. Common traumatic events that lead to PTSD include sexual assault and war experience. Negative childhood experiences are known to correlate with PTSD, including domestic ...
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