Pathology notes

Apr 16th, 2015
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Cellular AdaptationsDr. Tito,Dept. of Laboratory Medicine andPathologyHyperplasia Increase in the number of cells in anorgan or tissue May or may not be seen together withhypertrophy Can be either physiologic or pathologicPhysiologic Hyperplasia Hormonal Hyperplasia of uterine muscle duringpregnancy Compensatory Hyperplasia in an organ after partialresection Mechanisms include increased DNAsynthesis Growth inhibitors will halt hyperplasiaafter sufficient growth has occurredPathologic Hyperplasia Due to excessive hormonal stimulation Endometrial proliferation due to increasedabsolute or relative amount of estrogen Due to excessive growth factorstimulation Warts arising from papillomaviruses Not in itself neoplastic or preneoplastic but the underlying trigger may put thepatient at increased risk for developingsequelae (e.g., dysplasia or carcinoma)Hypertrophy Increase in the size of cells leading toan increase in the size of the organ(often seen in tissues made up ofterminally differentiated cells they canno longer divide, their only responseto the stress is to enlarge) End result is that the amount ofincreased work that each individual cellmust perform is limited Can be either physiologic or pathologicHypertrophy (contd) Physiologic Due to hormonal stimulation (e.g.,hypertrophy of uterine smooth muscleduring pregnancy) Pathologic Due to c

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