Medicine an Surgery notes

Apr 24th, 2015
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Hydrops Fetalis : APathophysiologicPerspectiveDr. Raphi Pollack,Dept. of Obstetrics & Gynecology,Bikur Cholim Hospital,Jerusalem.Hydrops FetalisEtiology Physiologic principles The fetus - a unique model Experimental models of hydrops Clinical observations ConclusionsHydrops Fetalis : EtiologyImmune NonimmuneCongenital anomaliesFetal infectionsFetal anemiasMetabolic disordersChromosomal abnormalitiesPlacental causesHydrops FetalisFinal Common PathwayStarlings LawJv = CFC ( P - Starlings LawJv = CFC ( P Jv = fluid flux across capillarycolloid osmotic reflectioncoefficientCFC = capillary filtration coefficientP = net hydrostatic pressure gradientPathogenesis of HydropsSevere anemiaHepatic extramedullary hematopoeisisDecreased prdtn of plasma proteinsDecreased plasma COPPathogenesis of HydropsCongestive heart failureIncreased central venous pressureIncreased capillary hydrostatic pressurePathogenesis of HydropsSevere tissue hypoxiaEndothelial cell damageCapillary leak of fluid & proteinDecreasedCOPIncreasedCVPIncreasedfluid effluxfrom intravascularspaceCapillaryleakThe Fetal MicrocirculationIncreased capillary permeability to plasmaproteins. Fivefold increase in CFC wrt adult Increased compliance of interstitial spa

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