Academia de Studii Economice Facultatea de Relaţii Economice Internaţionale Masterul Managementul Af

Jun 21st, 2015
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Acadia University
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Lucrarea de faţă îşi propune să analizeze cum şi mai ales de ce a eşuat afacerea mileniului, în ciuda forţelor complementare ale celor două companii, care le-au determinat iniţial să construiască o relaţie ce se dorea a fi mutual benefică.

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MEDICAL SURGICAL Abby TabuenaOverview of the Structures & Functions of Nervous SystemCentral NSPNSANSBrain & spinal cord 31 spinal & cranial sympathetic NS Parasypathatic NS Somatic NSC- 8T- 12L- 5S- 5C- 1ANS (or adrenergic of parasympatholitic response) SNS involved in fight or aggression responseEffects of SNS (anti-cholinergic/adrenergic)1. Dilate pupil to aware of surroundingsRelease of norepinephrine (adrenaline cathecolamine) - medriasisAdrenal medulla (potent vasoconstrictor)2. Dry mouthIncreases body activitiesVS = Increase3. BP & HR= increasedExcept GIT decrease GITmotility bronchioles dilated to take more oxygen4. RR increased* Why GIT is not increased = GIT is not important!5. Constipation & urinary retentionIncrease blood flow to skeletal muscles, brain & heart.I. Adrenergic Agents Epinephrine (adrenaline)SE: SNS effectII. PNS: Beta adrenergic blocking agents (opposite of adrenergic agents) (all end in lol)Blocks release of norepinephrine.Decrease body activities except GIT (diarrhea) Ex. Propanolol, MetopanololSE: B broncho spasm (bronchoconstriction)E elicits a decrease in myocardial contractionT treats HPNA AV conduction slows downGiven to angina & MI beta-blockers to rest heart Anti HPN agents:1. Beta blockers (-lol)2. Ace inhibitors (-pril) ex ENALAPR

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